The Symptoms

Progressively worsening (in some cases) symptoms Blurred Vision, Speech Problems, Fatigue, Muscle Weakness, Poor Co-ordination, Walking Difficulties.

What causes it?

A Dysfunctional Leaky digestive system leading to a dysfunctional auto immune system exacerbated by other factors including a diet with too many starchy foods (breads, pastry, biscuits, breakfast cereals and high starch root vegetables).

Stress, worry, pharmaceutical drugs (Antibiotics, Steroids, Ibuprofen, Aspirin etc., Toxic inorganic metals such as Mercury, Viral, Bacterial Problems.

The Effect

Your body’s own immune system attacks the myelin sheath, destroying patchy areas. This leads to scar tissue forming that in turn blocks or slows the nerve signals.

The Solution

Clear away the scar tissue, restore a healthy digestive system and to provide all of the nutrients to repair the damaged tissue.

How can Serrapeptase Help?

Serrapeptase 80,000IU with MSM and Trace Minerals helps immensely, as it clears out the inflammation and dead/scar tissue. By clearing this problem tissue, it enables the nerve signal to work unimpeded and possibly the body’s own healing system to replace it with healthy tissue.

How many do I take?

Start with 2 Serrapeptase 80,000IU Tablets/Capsules with MSM and Trace Minerals x 3 times per day on an empty stomach and increase it to 4 x 3 if no relief within 7 days. Then gradually reduce to 1 x 1.

Can I take too many tablets or can it interfere with any drugs I am taking?

No. It has been used for over 25 years with no side effects reported.

There are no studies that I know of using Serrapeptase with MS. You can see the available type of studies below. I found it useful for MS by chance and at first was puzzled why it could help. Serrapeptase has been sold as an anti-inflammatory for about 30 years in Europe and has also been found to be effective at dissolving non-living tissue and especially internal scarring and lesions. My hypothesis is that as the problem with MS is the scar tissue that forms on the damaged myelin sheath, then the Serrapeptase is dissolving this and allowing the nerves to start to function.

If there is no damaging attack on the myelin sheaf at that point, then it may even start to regenerate (although I do not have any evidence of this). I can only go by the good remission reports from the users (even with those with non-remissive type). The users are also using CurcuminX4000 (extract of turmeric).
There are no confirmed contraindications in 30 years of use. It is an OTC product and is already widely available in European countries.

NEW ORLEANS (Reuters Health) – Preliminary studies in rats suggest that curcumin, a compound found in the curry spice turmeric, may block the progression of multiple sclerosis (MS).

According to researcher Dr. Chandramohan Natarajan of Vanderbilt University in Nashville, Tennessee, rats with an MS-like illness showed little or no signs of disease symptoms after being injected with curcumin, while animals without the treatment went on to severe paralysis.

“We got a very good inhibition of the disease by treating with curcumin,” Natarajan told Reuters Health. He presented the findings at the annual Experimental Biology 2002 conference. No one knows what causes multiple sclerosis, in which the body’s immune system attacks the protective myelin sheath surrounding nerve fibres in the brain and spine. Symptoms of multiple sclerosis include muscle weakness and stiffness, balance and coordination problems, numbness and vision disturbances. Interest in the potential neuroprotective properties of curcumin arose after studies found very low levels of neurological diseases, such as Alzheimer’s, in elderly Indian populations. Added to this were studies confirming curcumin as a potent antiinflammatory agent, effective in wound healing. And just last fall, researchers at the University of California, Los Angeles, reported that curcumin appeared to slow the progression of Alzheimer’s in mice.
In their 30-day study, Natarajan and co-researcher Dr. John Bright gave injections of 50- and 100-microgram doses of curcumin, three times per week, to a group of mice bred to develop a disease called experimental autoimmune encephalomyelitis (EAE)–an autoimmune condition used by researchers as a model for multiple sclerosis because it also results in the slow erosion of myelin. They then watched the rats for signs of MS-like neurological impairment. By day 15, rats who had not received curcumin developed EAE to such an extent that they displayed complete paralysis of both hind limbs, according to Natarajan.
In contrast, rats given the 50-microgram dose of the curry compound showed only minor symptoms, such as a temporarily stiff tail. And rats given the 100-microgram dose appeared completely unimpaired throughout the 30 days of the study. The results didn’t really surprise Natarajan. “In Asian countries, such as India, China, who are eating more spicy foods, more yellow compounds like curcumin… there are only very, very rare reports of MS,” he pointed out. He said the doses the rats received were roughly equivalent in human terms to those found in a typical Indian diet.

Just how curcumin might work to thwart the progression of demyelinization remains unclear. But the Nashville researchers believe it may interrupt the
production of IL-12, a protein that plays a key role in signalling immune cells to launch their assault on the myelin sheath. Natarajan stressed that “we have to do a lot of work on this,” including examining other potential mechanisms by which curcumin slows EAE and, potentially, MS. The work remains preliminary, and MS patients should follow their doctor’s advice when it comes to treating the disease. Still, Natarajan said adding a little curry to the diet couldn’t hurt. “I think using this spice in their food could be of help,” he said.

Further Research

Dr. Hector E. Solorzano del Rio, M.D., Ph.D., D.Sc., chairman of the Program for Studies of Alternative Medicines of the University of Guadalajara, claims it has been scientifically proven that enzymes indeed help MS patients.

Dr. Solorzano gives an example about one of his wheelchair-bound MS patients, named Jose. At 40 years of age, he had received all known orthodox treatments
with no results. After one month on an enzyme program, Jose felt more strength in all of his muscles. He could again dress himself; within three months he could walk with some difficulty. After six months of enzymatic treatment, he was no longer sick. He is very happy and now lives a productive life. Important additional factors found in MS patients are a deficiency of unsaturated fatty acids and other essential nutrients. Hundreds of patients have been treated
with enzymatic therapy, with good results, not only in Germany or Mexico, but also in many other countries. It is important to remember that these patients also had a full dietary program.

What things can I do to help with MS?

1. Follow as below (in order of priority, as much as you can afford)

• Take Serrapeptase Enzymes as recommended

• Take CurcuminX4000 as recommended, (Stimulates Glutathione) to protect the myelin sheath during regeneration.

• Treatment with HealthPoint, an electro-acupressure device has proved to be highly effective in treating MS problems. The HealthPoint unit simply
  stimulates the body’s own healing system, and it is effective over a vast range of MS-related problems. (see www.DoveHealth.com)

• Sublingual Homocysteine Control Formula

• Using the Basic Health Plan will ensure appropriate nutrients and actions are taken to help the healing process

• Exercise by walking fast for 60 minutes, at least 3 times per week.

• Vitamin D 4000iu daily

• Vitamin E – Build up to 1200iu per day with meals.

• Hemp Oil needed to regenerate myelin sheath.

• Active Life Liquid Formula

• Get more oxygen in your cells by taking Oxygen Promoting Enzymes or Ozone Therapy or Hyperbaric and eventually exercise by walking fast for 60 minutes at least 5 times per week.

Critical to restore the Digestive Tract.

For your Digestive Tract Recovery take (in order of priority):

• Essential Herbal Cleanse to cleanse and restore digestive system and whole body system (optional)

• Probiotic14 – Ensures a healthy digestive tract

• L-Glutamine – take 5 gms daily to heal the damaged digestive tract for 1-2 months.

• OxyPlus+ (Oxygen and Aloe Vera) -Take 1/2oz twice per day to help heal the digestive tract and support the Probiotic.

• Digestive Enzymes – Ensures proper digestion of food.

Serrapeptase References:

l.. Kee WH. Tan SL, Lee V. Salmon YM. The treatment of breast engorgement with Serrapeptase (Danzen): a random ized double-blind controlled trial. Singapore Med J. 1989:30(1):48-54.

2. M izukoshi, D. et al. A double-blind clinical study of serrapeptase in the treatment of chronic sinusitis. Igaku Ayrni 109:50-62.1979.

3. Carratu, L. et al. Physio-chemical and rheological research on mucolytic activity of serrapeptase in chronic broncho-pneumopathies. Curr. Ther. Res. 28(6):937-951. 1980.

4. Braga, P.C. et al. Effects of serrapeptase on muco-ciliary clearance in patients with chronic bronchitis. Curr. Ther. Res. 29(5):738-744,1981.

5. Mazzonie, A. et al. Evaluation of serrapeptase in acute or chronic inflammation of otorhinolaryngology pathology: a multicentre, double-blind randomized trial versus placebo. J. int. Med. Res. 18(5):379-388,1990.

6. Kakinumu, A. et al. Regression of fibrinolysis in scalded rats by administration of serrapeptase. Biochem. Pharmacol. 31:2861-2866,1982.

7. Marly, M. Enzymotherapie anti-inflammatoire a l’aide de la serrapeptase: resultats cliniques en traumatologie et en ORL. C RTherapeut. 3:9-19,1985.

8. Odagiri, J. et al. Clinical applications of serrapeptase in sinusitis. Med. Consult. New Remedy 6:201-209, 1979.

9. Yamazaki, J. et al. Anti-inflammatory activity of TSP, a protease produced by a strain of Serratia. Folia Pharmacol. Japon. 6^302-314,1967.

I0. Harad~, Y. Clinical efficacy of serrapeptase on buccal swelling after radical operation for chronic sinusitis. Igaku Ayumi 123:768-778.1982.

1 I. Matsudo, A. et at. Effect of serrapeptase (Danzen) on inflammatory edema following operation for thyropid disease. Med. Consult. New Remedy 18:171-175, 1981.

12. Fujitani, T. et al. Effect of anti-inflammatory agent on transfer of antibiotics to the maxillary sinus mucosa in chronic sinusitis. Otorhinolaryngol. Clin. North Am. 66:557-565. 1976.

13. Tago. T. and Mitsui, S. Effects of serrapeptase in dissolution of sputum, especially in patients with bronchial asthma. Jap. Clin. Exp. Med. 49:222-228, 1972.

14. Mazzonie, A. et al. Evaluation of serrapeptase in acute or chronic inflammation of otorhinolaryngology pathology: a multicentre, double-blind randomized trial versus placebo. J. int. Med. Res. 18(5):379-388,1990.

15. Kase, Y. et al. A new method for evaluating mucolytic expectorant activity and its application. II. Application to two proteolytic enzymes, serrapeptase and seaprose. Arzneimittelforschung 32:374-378,1982.

16. Marriott, C. Modification of the rheoloaical properties of mucus by drugs. Adv. Exp. Med. Biol. 144^75-84, 1982.

17. Majima. Y. et al. Effects of orally administered drugs on dynamic viscoelasticity of human nasal mucus. Am. Rev. Respit. Dis. 141:79-83.1990.

18. Miyata, K. Intestinal absorption of serrapeptase. J ApplBiochem. 1980:2:111-16.

19. Aso T. et al. Breast engorgement and its treatment: Clinical effects of Danzen (serrapeptase) an anti-inflammatory enzyme preparation. The world of Obstetrics and Gynecology (Japanese). 1981:33:371-9.

20. Esch PM, Gemgross H. Fabian A. Reduction of postoperative swelling. Objective measurement of swelling of the upper ankle joint in treatment with serrapeptase-a prospective study (German). FortschrMed. 1989; 107(4):67-8, 71-2.

21. Majima Y, lnagaki M, Hirata K. Takeuchi K, M orishita A, Sakakura Y. The effect of an orally administered proteolytic enzyme on the elasticity and viscosity of nasal mucus. Arch Otorhinolaryngol. 1988;244(6):355-9.

22. Selan L, Berlutti F, Passariello C. Comodi-Ballanti MR, Thaller MC. proteolytic enzymes: a new treatment strategy for prosthetic infections? Antimicrob Agents Cheroother. 1993; 37(12):2618-21.

23. Koyama A, Mori J, Tokuda H, Waku M, Anno H, Katayama T, Murakami K, Komatsu H, Hirata M, Arai T, et al. Augmentation by serrapeptase of tissue permeation by cefotiam (Japanese). Jpn JAntibiot. 1986; 39(3):761-71.